Epigenetic repression of the bone morphogenetic protein type II receptor expression in scleroderma endothelial cells: a possible basis for enhanced SSc endothelial vulnerability to apoptosis
Oxidant stress is essential for the expression of TGF-beta fibrotic effects.
Sequence-specific knockdown of FLI1 gene by stealth small interfering RNA results in upregulation of matrix protein expression and the generation of activated fibroblast phenotype.
