Prenatal alcohol exposure causes long-term serotonin neuron deficit in mice Article (Faculty180)

cited authors

  • Sari, Youssef; Zhou, Feng C


  • Previous work from this laboratory showed that prenatal alcohol exposure at approximately 100 mg/dl from embryonic day (E)7 to early midgestation reduced the number and retarded the migration of serotonin (5-HT) neurons in the raphe nuclei in C57BL/6 mice. In this study, we report that the deficit of 5-HT neurons found in midgestation persisted on E18 and into young adulthood.


publication date

  • 2004

start page

  • 941

end page

  • 8


  • 28