Candida albicans
is a pervasive commensal fungus that is the most common pathogen responsible for invasive fungal infection (IFI). With incidence of IFI on the rise due to increasing susceptible populations, it is imperative that we investigate how
Candida albicans
interacts with blood components. When stimulating either human or mouse whole blood with thrombin, we saw a significant decrease in
C. albicans
survival. We then repeated
Candida
killing assays with thrombin-stimulated or unstimulated washed platelets and saw a similar decrease in CFU.
